The Wernicke area

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منابع مشابه

Wernicke ’ S Encephalopathy Revisited

Aims: A translation into English of the case history section of Carl Wernicke’s original manuscript of 1881, with a discussion on its relevance for clinicians today. Methods: A copy of Carl Wernicke’s original German text was obtained by one of the authors (CCHC) and translated into English from the old German by a professional translator. Results: The translation was subsequently agreed by nat...

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Wernicke-Korsakoff syndrome

Wernicke-Korsakoff syndrome is a manifestation of thiamine (vitamin B1) deficiency, usually secondary to alcohol abuse, although there other non-alcohol related causes, such as bariatric surgery, and extreme dietary restriction. The syndrome is a combined manifestation of the short-lived but acute Wernicke’s encephalopathy (WE), followed by chronic Korsakoff syndrome. It is generally accepted t...

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Wernicke Korsakoff Encephalopathy

Carl Wernicke described first Wernicke encephalopathy in 1881. He reported a pathological clinical situation, which comprises a triad of symptoms with acute mental confusion, ataxia, and ophthalmoplegia. He described three patients, two with a story of high alcohol consumption and one patient with persistent vomiting that presented with progressive stupor and coma, progressing to death. After t...

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Wernicke-Korsakoff encephalopathy and the endorphin system.

Sir, We read with interest the recent paper of Gibb et al. (1985) in which reversible coma in Wernicke's encephalopathy was reported. Treatment with thiamine was successful in that the patient regained consciousness but unsuccessful in preventing the residual amnestic defect of Korsakoff's encephalopathy. In a similar report (Wallis et al., 1978) four comatose patients with the Wernicke syndrom...

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Neuroimaging of the Wernicke-Korsakoff syndrome.

AIM Presented is the neuroradiological signature of acute Wernicke's encephalopathy (WE), derived from different types of magnetic resonance imaging (MRI) sequences. WE results from thiamine depletion, and its most typical antecedent is chronic alcohol dependence. Brain regions observed with in vivo MRI affected in acute WE include the mammillary bodies, periaqueductal and periventricular gray ...

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ژورنال

عنوان ژورنال: Neurology

سال: 2015

ISSN: 0028-3878,1526-632X

DOI: 10.1212/wnl.0000000000002219